Therosclerosis Vi
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Atherosclerosis VI
Author: F. G. Schettler
language: en
Publisher: Springer Science & Business Media
Release Date: 2012-12-06
In 1982 Berlin was host for the second time to the International Symposium on Atherosclerosis. In 1973 the third symposium was held there, following the first in Athens -opened by the unforgettable Paul D. White -and the second in Chicago, where the great gentlemen of atherosclerosis research - Louis Katz and Irving Page - left their special imprint on the meeting. Since the third symposium in Berlin impressive advances have been made in the field of atherosclerosis. The symposia in Tokyo in 1976 and in Houston in 1979 introduced important new knowledge from current research, stimulating worldwide interest; Berlin highlighted the latest developments. The International Atherosclerosis Society (lAS) provides an international forum for the entire field of atherosclerosis research. Its main purpose is to apply the results of basic research to clinical medicine, and thereby to benefit the practitioner. Prevention and rehabilitation are of special importance. Intensified international cooperation is urgently needed at all levels. A declared goal of the lAS is contact between young investigators and between international research and work groups. Participating in the Berlin meeting were 1400 researchers from 42 nations. This illustrates the growing interest in atherosclerosis as the leading cause of death in "developed" societies. However, the incidence of atherosclerosis is increasing worldwide, although there are national differences in the pattern and appearance of the disease; this was documented at the Berlin Symposium.
Atherosclerosis VI
This volume is the sixth in the Saratoga International Conference series, which brings together leading researchers and clinicians from Asia, Europe, and North America to highlight the latest findings on the genetics, development, prognosis, and the pathologies that lead to atherosclerosis. Papers describe recent research in vascular biology; signal transduction and angiogenesis; inflammation, thrombosis and atherosclerosis; lipids, lipoproteins, and their receptors; lipid metabolism and inflammation; genetic approaches to atherosclerosis; and coronary artery disease and its therapy.
Regression of Atherosclerotic Lesions
Author: M. Rene Malinow
language: en
Publisher: Springer Science & Business Media
Release Date: 2013-06-29
The Advanced Study Institute on Regression of Atherosclerotic Lesions, sponsored by the North Atlantic Treaty Organization, was held in Maratea, Italy, September 11-13, 1983. The institute was designed to foster discussion of experimental data on the regression of atherosclerosis in animals and evidence of a similar phenomenon in humans. The participating scientists showed with anatomical and biochemical data, that atherosclerosis regresses in cholesterol fed animals after withdrawal of atherogenic stimuli. The existence of regression in humans was inferred from sequential angiographic visualization of arteries, but anatomical and biochemical characteristics of the process have not been defined. Two mechanisms are probably involved in such an angiographic regression. The first, which may occur when hyperlipidemia is partly or fully corrected or during undernutrition or other metabolic abnormalities, may be related to atherosclerosis changes noted in animals undergoing regression. The second type seems independent of changes in plasma lipid levels and may involve resolution of thrombi or other underlying arterial wall factors. The relative frequencies of the two mechanisms are unknown; whether they can occur simultaneously also warrants consideration. It is likely that an exciting milestone will be forthcoming when regression of atherosclerosis is unequivocally demonstrated in humans. Publication of these institute proceedings constitutes a valuable background for future developments.